abnormal liver physiology_Insr<tm1Shs>/Insr<+>

http://metadb.riken.jp/0000609IC57BL6CrSlcInsrtm1ShsInsrInsrInsrtm1Shs

abnormal liver physiology_Insr<tm1Shs>/Insr<+>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• insulin receptor
  http://metadb.riken.jp/db/mgi_rdf/MGI:96575

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Insr<tm1Shs>/Insr<+>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Insr<+>|Insr<tm1Shs>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: C57BL/6CrSlc

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal liver physiology
  http://purl.obolibrary.org/obo/MP_0000609

文献

http://purl.org/net/cito/citesAsAuthority

• Tomonori. Baba, et.al., Estrogen, insulin, and dietary signals cooperatively regulate longevity signals to enhance resistance to oxidative stress in mice., The Journal of biological chemistry, 2005, 280
  http://rdf.ncbi.nlm.nih.gov/pubmed/15713666