abnormal cardiovascular system physiology_Nos3<tm1Unc>/Nos3<tm1Unc>

http://metadb.riken.jp/0001544I129P2OlaHsdC57BL6Nos3tm1UncNos3tm1UncNos3tm1Unc

abnormal cardiovascular system physiology_Nos3<tm1Unc>/Nos3<tm1Unc>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• nitric oxide synthase 3, endothelial cell
  http://metadb.riken.jp/db/mgi_rdf/MGI:97362

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Nos3<tm1Unc>/Nos3<tm1Unc>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Nos3<tm1Unc>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129P2/OlaHsd * C57BL/6

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal cardiovascular system physiology
  http://purl.obolibrary.org/obo/MP_0001544

文献

http://purl.org/net/cito/citesAsAuthority

• Alyson A. Miller, et.al., Deletion of the eNOS gene has a greater impact on the pulmonary circulation of male than female mice., American journal of physiology. Lung cellular and molecular physiology, 2005, 289
  http://rdf.ncbi.nlm.nih.gov/pubmed/15821017