increased inflammatory response_Fcgr2b<tm1Ttk>/Fcgr2b<tm1Ttk>

http://metadb.riken.jp/0001846B6129S4Fcgr2btm1TtkFcgr2btm1TtkFcgr2btm1TtkFcgr2btm1Ttk

increased inflammatory response_Fcgr2b<tm1Ttk>/Fcgr2b<tm1Ttk>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• Fc receptor, IgG, low affinity IIb
  http://metadb.riken.jp/db/mgi_rdf/MGI:95499

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Fcgr2b<tm1Ttk>/Fcgr2b<tm1Ttk>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Fcgr2b<tm1Ttk>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6.129S4-Fcgr2b<tm1Ttk>

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• increased inflammatory response
  http://purl.obolibrary.org/obo/MP_0001846

文献

http://purl.org/net/cito/citesAsAuthority

• S. Bolland, et.al., Spontaneous autoimmune disease in Fc(gamma)RIIB-deficient mice results from strain-specific epistasis., Immunity, 2000, 13
  http://rdf.ncbi.nlm.nih.gov/pubmed/10981970