abnormal kidney physiology_Fas<lpr>/Fas<lpr>

http://metadb.riken.jp/0002136MRLMpJFaslprJFaslprFaslprFaslpr

abnormal kidney physiology_Fas<lpr>/Fas<lpr>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• Fas (TNF receptor superfamily member 6)
  http://metadb.riken.jp/db/mgi_rdf/MGI:95484

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Fas<lpr>/Fas<lpr>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Fas<lpr>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• MRL/MpJ-Fas<lpr>/J

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal kidney physiology
  http://purl.obolibrary.org/obo/MP_0002136

文献

http://purl.org/net/cito/citesAsAuthority

• Xiaodong. He, et.al., Deficiency of P-selectin or P-selectin glycoprotein ligand-1 leads to accelerated development of glomerulonephritis and increased expression of CC chemokine ligand 2 in lupus-prone mice., Journal of immunology (Baltimore, Md. : 1950), 2006, 177
  http://rdf.ncbi.nlm.nih.gov/pubmed/17142777