no abnormal phenotype detected_Smad7<tm1Gkh>/Smad7<tm1Gkh>

http://metadb.riken.jp/0002169B6N129S6Smad7tm1GkhSmad7tm1GkhSmad7tm1GkhSmad7tm1Gkh

no abnormal phenotype detected_Smad7<tm1Gkh>/Smad7<tm1Gkh>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• SMAD family member 7
  http://metadb.riken.jp/db/mgi_rdf/MGI:1100518

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Smad7<tm1Gkh>/Smad7<tm1Gkh>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Smad7<tm1Gkh>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6N.129S6-Smad7<tm1Gkh>

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Anton. Gisterå, et.al., Transforming growth factor-β signaling in T cells promotes stabilization of atherosclerotic plaques through an interleukin-17-dependent pathway., Science translational medicine, 2013, 5
  http://rdf.ncbi.nlm.nih.gov/pubmed/23903754