no abnormal phenotype detected_Heyl<tm1Gess>/Heyl<tm1Gess>

http://metadb.riken.jp/0002169EB6129P2Heyltm1GessorI129P2OlaHsdC57BL6Heyltm1GessHeyltm1GessHeyltm1Gess

no abnormal phenotype detected_Heyl<tm1Gess>/Heyl<tm1Gess>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• hairy/enhancer-of-split related with YRPW motif-like
  http://metadb.riken.jp/db/mgi_rdf/MGI:1860511

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Heyl<tm1Gess>/Heyl<tm1Gess>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Heyl<tm1Gess>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• either: B6.129P2-Heyl<tm1Gess> or (involves: 129P2/OlaHsd * C57BL/6)

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Andreas. Fischer, et.al., Combined loss of Hey1 and HeyL causes congenital heart defects because of impaired epithelial to mesenchymal transition., Circulation research, 2007, 100
  http://rdf.ncbi.nlm.nih.gov/pubmed/17303760