no abnormal phenotype detected_Efnb3<tm1.1Henk>/Efnb3<tm1.1Henk>

http://metadb.riken.jp/0002169EI129S1Sv129X1SvJorI129S1Sv129X1SvJC57BL6orI129S1Sv129X1SvJCD1Efnb3tm11HenkEfnb3tm11HenkEfnb3tm11Henk

no abnormal phenotype detected_Efnb3<tm1.1Henk>/Efnb3<tm1.1Henk>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• ephrin B3
  http://metadb.riken.jp/db/mgi_rdf/MGI:109196

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Efnb3<tm1.1Henk>/Efnb3<tm1.1Henk>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Efnb3<tm1.1Henk>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• either: (involves: 129S1/Sv * 129X1/SvJ) or (involves: 129S1/Sv * 129X1/SvJ * C57BL/6) or (involves: 129S1/Sv * 129X1/SvJ * CD-1)

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• N. Yokoyama, et.al., Forward signaling mediated by ephrin-B3 prevents contralateral corticospinal axons from recrossing the spinal cord midline., Neuron, 2001, 29
  http://rdf.ncbi.nlm.nih.gov/pubmed/11182083