no abnormal phenotype detected_Apc<tm1Rsmi>/Apc<tm1Rsmi>

http://metadb.riken.jp/0002169I129P2OlaHsdApctm1RsmiApctm1RsmiApctm1Rsmi

no abnormal phenotype detected_Apc<tm1Rsmi>/Apc<tm1Rsmi>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• adenomatosis polyposis coli
  http://metadb.riken.jp/db/mgi_rdf/MGI:88039

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Apc<tm1Rsmi>/Apc<tm1Rsmi>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Apc<tm1Rsmi>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129P2/OlaHsd

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Razvan L. Miclea, et.al., Adenomatous polyposis coli-mediated control of beta-catenin is essential for both chondrogenic and osteogenic differentiation of skeletal precursors., BMC developmental biology, 2009, 9
  http://rdf.ncbi.nlm.nih.gov/pubmed/19356224