no abnormal phenotype detected_Slc22a2<tm1Ahs>/Slc22a2<tm1Ahs>

http://metadb.riken.jp/0002169I129P2OlaHsdFVBSlc22a2tm1AhsSlc22a2tm1AhsSlc22a2tm1Ahs

no abnormal phenotype detected_Slc22a2<tm1Ahs>/Slc22a2<tm1Ahs>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• solute carrier family 22 (organic cation transporter), member 2
  http://metadb.riken.jp/db/mgi_rdf/MGI:1335072

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Slc22a2<tm1Ahs>/Slc22a2<tm1Ahs>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Slc22a2<tm1Ahs>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129P2/OlaHsd * FVB

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Johan W. Jonker, et.al., Deficiency in the organic cation transporters 1 and 2 (Oct1/Oct2 [Slc22a1/Slc22a2]) in mice abolishes renal secretion of organic cations., Molecular and cellular biology, 2003, 23
  http://rdf.ncbi.nlm.nih.gov/pubmed/14560032