no abnormal phenotype detected_Nlgn3<tm2.1Sud>/Y

http://metadb.riken.jp/0002169I129S1Sv129X1SvJC57BL6Nlgn3tm21SudYNlgn3tm21Sud

no abnormal phenotype detected_Nlgn3<tm2.1Sud>/Y

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• neuroligin 3
  http://metadb.riken.jp/db/mgi_rdf/MGI:2444609

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Nlgn3<tm2.1Sud>/Y

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Nlgn3<tm2.1Sud>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129S1/Sv * 129X1/SvJ * C57BL/6

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Katsuhiko. Tabuchi, et.al., A neuroligin-3 mutation implicated in autism increases inhibitory synaptic transmission in mice., Science (New York, N.Y.), 2007, 318
  http://rdf.ncbi.nlm.nih.gov/pubmed/17823315