no abnormal phenotype detected_Zbtb32<tm1Ppp>/Zbtb32<tm1Ppp>

http://metadb.riken.jp/0002169I129S1SvZbtb32tm1PppZbtb32tm1PppZbtb32tm1Ppp

no abnormal phenotype detected_Zbtb32<tm1Ppp>/Zbtb32<tm1Ppp>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• zinc finger and BTB domain containing 32
  http://metadb.riken.jp/db/mgi_rdf/MGI:1891838

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Zbtb32<tm1Ppp>/Zbtb32<tm1Ppp>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Zbtb32<tm1Ppp>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129S1/Sv

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• no abnormal phenotype detected
  http://purl.obolibrary.org/obo/MP_0002169

文献

http://purl.org/net/cito/citesAsAuthority

• Francesco. Piazza, et.al., Disruption of PLZP in mice leads to increased T-lymphocyte proliferation, cytokine production, and altered hematopoietic stem cell homeostasis., Molecular and cellular biology, 2004, 24
  http://rdf.ncbi.nlm.nih.gov/pubmed/15542853