abnormal T cell physiology_H2-Ab1<bm12>/H2-Ab1<bm12>

http://metadb.riken.jp/0002444B6CH2Ab1bm12KhEgJH2Ab1bm12H2Ab1bm12H2Ab1bm12

abnormal T cell physiology_H2-Ab1<bm12>/H2-Ab1<bm12>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• histocompatibility 2, class II antigen A, beta 1
  http://metadb.riken.jp/db/mgi_rdf/MGI:103070

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• H2-Ab1<bm12>/H2-Ab1<bm12>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• H2-Ab1<bm12>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6(C)-H2-Ab1<bm12>/KhEgJ

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal T cell physiology
  http://purl.obolibrary.org/obo/MP_0002444

文献

http://purl.org/net/cito/citesAsAuthority

• Mathilde A. Poussin, et.al., Suppressed clinical experimental autoimmune myasthenia gravis in bm12 mice is linked to reduced intracellular calcium mobilization and IL-10 and IFN-gamma release by acetylcholine receptor-specific T cells., Journal of neuroimmunology, 2003, 134
  http://rdf.ncbi.nlm.nih.gov/pubmed/12507777