abnormal prepulse inhibition_Drd2<tm1Low>/Drd2<tm1Low>

http://metadb.riken.jp/0003088B6129S2Drd2tm1LowDrd2tm1LowDrd2tm1LowDrd2tm1Low

abnormal prepulse inhibition_Drd2<tm1Low>/Drd2<tm1Low>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• dopamine receptor D2
  http://metadb.riken.jp/db/mgi_rdf/MGI:94924

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Drd2<tm1Low>/Drd2<tm1Low>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Drd2<tm1Low>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6.129S2-Drd2<tm1Low>

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal prepulse inhibition
  http://purl.obolibrary.org/obo/MP_0003088

文献

http://purl.org/net/cito/citesAsAuthority

• R J. Ralph, et.al., The dopamine D2, but not D3 or D4, receptor subtype is essential for the disruption of prepulse inhibition produced by amphetamine in mice., The Journal of neuroscience : the official journal of the Society for Neuroscience, 1999, 19
  http://rdf.ncbi.nlm.nih.gov/pubmed/10341260