abnormal response/metabolism to endogenous compounds_Cd209a<tm1.1Cfg>/Cd209a<tm1.1Cfg>

http://metadb.riken.jp/0003638B6FVBCd209atm11CfgMmucdCd209atm11CfgCd209atm11CfgCd209atm11Cfg

abnormal response/metabolism to endogenous compounds_Cd209a<tm1.1Cfg>/Cd209a<tm1.1Cfg>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• CD209a antigen
  http://metadb.riken.jp/db/mgi_rdf/MGI:2157942

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Cd209a<tm1.1Cfg>/Cd209a<tm1.1Cfg>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Cd209a<tm1.1Cfg>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6(FVB)-Cd209a<tm1.1Cfg>/Mmucd

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal response/metabolism to endogenous compounds
  http://purl.obolibrary.org/obo/MP_0003638

文献

http://purl.org/net/cito/citesAsAuthority

• Xin-Jiang. Lu, et.al., LECT2 protects mice against bacterial sepsis by activating macrophages via the CD209a receptor., The Journal of experimental medicine, 2013, 210
  http://rdf.ncbi.nlm.nih.gov/pubmed/23254286