abnormal response/metabolism to endogenous compounds_Agtr2<tm1Tin>/Y

http://metadb.riken.jp/0003638I129P2OlaHsdC57BL6Agtr2tm1TinYAgtr2tm1Tin

abnormal response/metabolism to endogenous compounds_Agtr2<tm1Tin>/Y

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• angiotensin II receptor, type 2
  http://metadb.riken.jp/db/mgi_rdf/MGI:87966

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Agtr2<tm1Tin>/Y

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Agtr2<tm1Tin>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129P2/OlaHsd * C57BL/6

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal response/metabolism to endogenous compounds
  http://purl.obolibrary.org/obo/MP_0003638

文献

http://purl.org/net/cito/citesAsAuthority

• Florian. Gembardt, et.al., Cardiovascular phenotype of mice lacking all three subtypes of angiotensin II receptors., FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2008, 22
  http://rdf.ncbi.nlm.nih.gov/pubmed/18497303