oxidative stress_Atm<tm1Awb>/Atm<tm1Awb>

http://metadb.riken.jp/0003674E129S6SvEvTacAtmtm1AwborI129S6SvEvTacNIHBlackSwissAtmtm1AwbAtmtm1AwbAtmtm1Awb

oxidative stress_Atm<tm1Awb>/Atm<tm1Awb>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• ataxia telangiectasia mutated
  http://metadb.riken.jp/db/mgi_rdf/MGI:107202

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Atm<tm1Awb>/Atm<tm1Awb>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Atm<tm1Awb>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• either: 129S6/SvEvTac-Atm<tm1Awb> or (involves: 129S6/SvEvTac * NIH Black Swiss)

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• oxidative stress
  http://purl.obolibrary.org/obo/MP_0003674

文献

http://purl.org/net/cito/citesAsAuthority

• C. Barlow, et.al., Loss of the ataxia-telangiectasia gene product causes oxidative damage in target organs., Proceedings of the National Academy of Sciences of the United States of America, 1999, 96
  http://rdf.ncbi.nlm.nih.gov/pubmed/10449794