abnormal lymphocyte physiology_Gpr132<tm1Witt>/Gpr132<tm1Witt>

http://metadb.riken.jp/0003945I129X1SvJBALBcGpr132tm1WittGpr132tm1WittGpr132tm1Witt

abnormal lymphocyte physiology_Gpr132<tm1Witt>/Gpr132<tm1Witt>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• G protein-coupled receptor 132
  http://metadb.riken.jp/db/mgi_rdf/MGI:1890220

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Gpr132<tm1Witt>/Gpr132<tm1Witt>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Gpr132<tm1Witt>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129X1/SvJ * BALB/c

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal lymphocyte physiology
  http://purl.obolibrary.org/obo/MP_0003945

文献

http://purl.org/net/cito/citesAsAuthority

• L Q. Le, et.al., Mice lacking the orphan G protein-coupled receptor G2A develop a late-onset autoimmune syndrome., Immunity, 2001, 14
  http://rdf.ncbi.nlm.nih.gov/pubmed/11371358