increased susceptibility to experimental autoimmune encephalomyelitis_H2-T23<tm3Cant>/H2-T23<tm3Cant>

http://metadb.riken.jp/0004799B6129S6H2T23tm3CantH2T23tm3CantH2T23tm3CantH2T23tm3Cant

increased susceptibility to experimental autoimmune encephalomyelitis_H2-T23<tm3Cant>/H2-T23<tm3Cant>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• histocompatibility 2, T region locus 23
  http://metadb.riken.jp/db/mgi_rdf/MGI:95957

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• H2-T23<tm3Cant>/H2-T23<tm3Cant>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• H2-T23<tm3Cant>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• B6.129S6-H2-T23<tm3Cant>

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• increased susceptibility to experimental autoimmune encephalomyelitis
  http://purl.obolibrary.org/obo/MP_0004799

文献

http://purl.org/net/cito/citesAsAuthority

• Linrong. Lu, et.al., Regulation of CD8+ regulatory T cells: Interruption of the NKG2A-Qa-1 interaction allows robust suppressive activity and resolution of autoimmune disease., Proceedings of the National Academy of Sciences of the United States of America, 2008, 105
  http://rdf.ncbi.nlm.nih.gov/pubmed/19047627