RDF of MGI data
表現型アノテーション
increased susceptibility to experimental autoimmune encephalomyelitis_Cd59a<tm1Bpm>/Cd59a<tm1Bpm>
http://metadb.riken.jp/0004799I129C57BL6Cd59atm1BpmCd59atm1BpmCd59atm1Bpm
increased susceptibility to experimental autoimmune encephalomyelitis_Cd59a<tm1Bpm>/Cd59a<tm1Bpm>(表現型アノテーション)
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increased susceptibility to experimental autoimmune encephalomyelitis_Cd59a<tm1Bpm>/Cd59a<tm1Bpm>
表現型アノテーション
MGI marker
http://metadb.riken.jp/db/mgi_rdf/variant_of
CD59a antigen
http://metadb.riken.jp/db/mgi_rdf/MGI:109177
遺伝子型
http://metadb.riken.jp/db/mgi_rdf/allele_composition
Cd59a<tm1Bpm>/Cd59a<tm1Bpm>
アレルシンボル
http://metadb.riken.jp/db/mgi_rdf/allele_symbol
Cd59a<tm1Bpm>
遺伝的背景
http://metadb.riken.jp/db/mgi_rdf/genetic_background
involves: 129 * C57BL/6
MP annotation
http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype
increased susceptibility to experimental autoimmune encephalomyelitis
http://purl.obolibrary.org/obo/MP_0004799
文献
http://purl.org/net/cito/citesAsAuthority
Richard James. Mead, et.al., Deficiency of the complement regulator CD59a enhances disease severity, demyelination and axonal injury in murine acute experimental allergic encephalomyelitis., Laboratory investigation; a journal of technical methods and pathology, 2004, 84
http://rdf.ncbi.nlm.nih.gov/pubmed/14631387
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