RDF of MGI data
表現型アノテーション
impaired glucose tolerance_Lepr<db>/Lepr<+>
http://metadb.riken.jp/0005293IC57BLKSJLeprdbLeprLeprdbLepr
impaired glucose tolerance_Lepr<db>/Lepr<+>(表現型アノテーション)
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impaired glucose tolerance_Lepr<db>/Lepr<+>
表現型アノテーション
MGI marker
http://metadb.riken.jp/db/mgi_rdf/variant_of
leptin receptor
http://metadb.riken.jp/db/mgi_rdf/MGI:104993
遺伝子型
http://metadb.riken.jp/db/mgi_rdf/allele_composition
Lepr<db>/Lepr<+>
アレルシンボル
http://metadb.riken.jp/db/mgi_rdf/allele_symbol
Lepr<db>|Lepr<+>
遺伝的背景
http://metadb.riken.jp/db/mgi_rdf/genetic_background
involves: C57BLKS/J
MP annotation
http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype
impaired glucose tolerance
http://purl.obolibrary.org/obo/MP_0005293
文献
http://purl.org/net/cito/citesAsAuthority
H. Yamashita, et.al., Leptin administration prevents spontaneous gestational diabetes in heterozygous Lepr(db/+) mice: effects on placental leptin and fetal growth., Endocrinology, 2001, 142
http://rdf.ncbi.nlm.nih.gov/pubmed/11416008
Raja. Nadif, et.al., The maternal environment programs postnatal weight gain and glucose tolerance of male offspring, but placental and fetal growth are determined by fetal genotype in the Leprdb/+ model of gestational diabetes., Endocrinology, 2015, 156
http://rdf.ncbi.nlm.nih.gov/pubmed/25353183
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