increased susceptibility to endotoxin shock_Mst1r<tm1.1Sew>/Mst1r<tm1.1Sew>

http://metadb.riken.jp/0008735I129OlaCD1NIHBlackSwissMst1rtm11SewMst1rtm11SewMst1rtm11Sew

increased susceptibility to endotoxin shock_Mst1r<tm1.1Sew>/Mst1r<tm1.1Sew>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• macrophage stimulating 1 receptor (c-met-related tyrosine kinase)
  http://metadb.riken.jp/db/mgi_rdf/MGI:99614

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Mst1r<tm1.1Sew>/Mst1r<tm1.1Sew>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Mst1r<tm1.1Sew>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129/Ola * CD-1 * NIH Black Swiss

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• increased susceptibility to endotoxin shock
  http://purl.obolibrary.org/obo/MP_0008735

文献

http://purl.org/net/cito/citesAsAuthority

• S E. Waltz, et.al., Ron-mediated cytoplasmic signaling is dispensable for viability but is required to limit inflammatory responses., The Journal of clinical investigation, 2001, 108
  http://rdf.ncbi.nlm.nih.gov/pubmed/11518730