abnormal pancreatic acinar cell physiology_Tg(Mt1-TGFBR2*)AM3Epb/0

http://metadb.riken.jp/0009147IFVBNTgMt1TGFBR2AM3Epb0TgMt1TGFBR2AM3Epb

abnormal pancreatic acinar cell physiology_Tg(Mt1-TGFBR2*)AM3Epb/0

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• transgene insertion AM3, Erwin P Bottinger
  http://metadb.riken.jp/db/mgi_rdf/MGI:5806464

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Tg(Mt1-TGFBR2*)AM3Epb/0

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Tg(Mt1-TGFBR2*)AM3Epb

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: FVB/N

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal pancreatic acinar cell physiology
  http://purl.obolibrary.org/obo/MP_0009147

文献

http://purl.org/net/cito/citesAsAuthority

• E P. Böttinger, et.al., Expression of a dominant-negative mutant TGF-beta type II receptor in transgenic mice reveals essential roles for TGF-beta in regulation of growth and differentiation in the exocrine pancreas., The EMBO journal, 1997, 16
  http://rdf.ncbi.nlm.nih.gov/pubmed/9184209