RDF of MGI data
pubmed
Matina. Economopoulou, et.al., Histone H2AX is integral to hypoxia-driven neovascularization., Nature medicine, 2009, 15
http://rdf.ncbi.nlm.nih.gov/pubmed/19377486
Matina. Economopoulou, et.al., Histone H2AX is integral to hypoxia-driven neovascularization., Nature medicine, 2009, 15(pubmed)
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Matina. Economopoulou, et.al., Histone H2AX is integral to hypoxia-driven neovascularization., Nature medicine, 2009, 15
pubmed
allele
http://purl.org/net/cito/citesAsAuthority
H2afx<tm2.1Fwa>
http://metadb.riken.jp/db/mgi_rdf/MGI:3849331
class
http://purl.org/net/cito/citesAsAuthority
H2afx<tm2.1Fwa>
http://metadb.riken.jp/db/mgi_rdf/MGI:3849331
表現型アノテーション
http://purl.org/net/cito/citesAsAuthority
decreased tumor growth/size_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0003447NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
retinal neovascularization_H2afx<tm1Nus>/H2afx<tm2.1Fwa>,Tg(Cdh5-cre)7Mlia/0
http://metadb.riken.jp/0008852I129S6SvEvTacFVBNH2afxtm1NusH2afxtm21FwaTgCdh5cre7Mlia0H2afxtm21FwaTgCdh5cre7MliaH2afxtm1Nus
abnormal tumor vascularization_H2afx<tm1Nus>/H2afx<tm2.1Fwa>,Tg(Cdh5-cre)7Mlia/0
http://metadb.riken.jp/0010144I129S6SvEvTacFVBNH2afxtm1NusH2afxtm21FwaTgCdh5cre7Mlia0H2afxtm21FwaTgCdh5cre7MliaH2afxtm1Nus
retinal neovascularization_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0008852NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
decreased cell proliferation_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0000352NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
abnormal physiological neovascularization_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0003710NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
increased vascular endothelial cell apoptosis_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0020321NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
abnormal tumor vascularization_H2afx<tm1Nus>/H2afx<tm1Nus>
http://metadb.riken.jp/0010144NSH2afxtm1NusH2afxtm1NusH2afxtm1Nus
decreased tumor growth/size_H2afx<tm1Nus>/H2afx<tm2.1Fwa>,Tg(Cdh5-cre)7Mlia/0
http://metadb.riken.jp/0003447I129S6SvEvTacFVBNH2afxtm1NusH2afxtm21FwaTgCdh5cre7Mlia0H2afxtm21FwaTgCdh5cre7MliaH2afxtm1Nus
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