André. Kleinridders, et.al., MyD88 signaling in the CNS is required for development of fatty acid-induced leptin resistance and diet-induced obesity., Cell metabolism, 2009, 10

http://rdf.ncbi.nlm.nih.gov/pubmed/19808018

André. Kleinridders, et.al., MyD88 signaling in the CNS is required for development of fatty acid-induced leptin resistance and diet-induced obesity., Cell metabolism, 2009, 10

pubmed

allele

http://purl.org/net/cito/citesAsAuthority

• Myd88<tm1Jcbr>
  http://metadb.riken.jp/db/mgi_rdf/MGI:4367063

class

http://purl.org/net/cito/citesAsAuthority

• Myd88<tm1Jcbr>
  http://metadb.riken.jp/db/mgi_rdf/MGI:4367063

表現型アノテーション

http://purl.org/net/cito/citesAsAuthority

• decreased susceptibility to diet-induced obesity_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0005659B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased body length_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0001258B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased epididymal fat pad weight_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0009289B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased food intake_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0011940B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• abnormal glucose homeostasis_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0002078B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased fat cell size_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0009269B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• hypoactivity_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0001402B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• increased energy expenditure_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0004889B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• abnormal parametrial fat pad morphology_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0008906B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased total body fat amount_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0010025B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• decreased circulating insulin level_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0002727B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln
• increased insulin sensitivity_Myd88<tm1Jcbr>/Myd88<tm1Jcbr>,Tg(Nes-cre)1Kln/0
  http://metadb.riken.jp/0002891B6CgMyd88tm1JcbrTgNescre1KlnMyd88tm1JcbrMyd88tm1JcbrTgNescre1Kln0Myd88tm1JcbrTgNescre1Kln