abnormal cell physiology_Gnl3<tm2.1Rylt>/Gnl3<tm2.1Rylt>,Trp53<tm1Tyj>/Trp53<tm1Tyj>,Tg(CAG-cre/Esr1*)5Amc/0

http://metadb.riken.jp/0005621I129129S2SvPasC57BL6CBAGnl3tm21RyltGnl3tm21RyltTrp53tm1TyjTrp53tm1TyjTgCAGcreEsr15Amc0Trp53tm1TyjTgCAGcreEsr15AmcGnl3tm21Rylt

abnormal cell physiology_Gnl3<tm2.1Rylt>/Gnl3<tm2.1Rylt>,Trp53<tm1Tyj>/Trp53<tm1Tyj>,Tg(CAG-cre/Esr1*)5Amc/0

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• guanine nucleotide binding protein-like 3 (nucleolar)
  http://metadb.riken.jp/db/mgi_rdf/MGI:1353651
• transgene insertion 5, Andrew P McMahon
  http://metadb.riken.jp/db/mgi_rdf/MGI:2182765
• transformation related protein 53
  http://metadb.riken.jp/db/mgi_rdf/MGI:98834

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Gnl3<tm2.1Rylt>/Gnl3<tm2.1Rylt>,Trp53<tm1Tyj>/Trp53<tm1Tyj>,Tg(CAG-cre/Esr1*)5Amc/0

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Trp53<tm1Tyj>|Tg(CAG-cre/Esr1*)5Amc|Gnl3<tm2.1Rylt>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129 * 129S2/SvPas * C57BL/6 * CBA

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• abnormal cell physiology
  http://purl.obolibrary.org/obo/MP_0005621

文献

http://purl.org/net/cito/citesAsAuthority

• Lingjun. Meng, et.al., Nucleostemin deletion reveals an essential mechanism that maintains the genomic stability of stem and progenitor cells., Proceedings of the National Academy of Sciences of the United States of America, 2013, 110
  http://rdf.ncbi.nlm.nih.gov/pubmed/23798389