increased sensitivity to induced morbidity/mortality_Fah<tm1Mgo>/Fah<tm1Mgo>

http://metadb.riken.jp/0009763I129S7SvEvBrdC57BL6Fahtm1MgoFahtm1MgoFahtm1Mgo

increased sensitivity to induced morbidity/mortality_Fah<tm1Mgo>/Fah<tm1Mgo>

表現型アノテーション

MGI marker

http://metadb.riken.jp/db/mgi_rdf/variant_of

• fumarylacetoacetate hydrolase
  http://metadb.riken.jp/db/mgi_rdf/MGI:95482

遺伝子型

http://metadb.riken.jp/db/mgi_rdf/allele_composition

• Fah<tm1Mgo>/Fah<tm1Mgo>

アレルシンボル

http://metadb.riken.jp/db/mgi_rdf/allele_symbol

• Fah<tm1Mgo>

遺伝的背景

http://metadb.riken.jp/db/mgi_rdf/genetic_background

• involves: 129S7/SvEvBrd * C57BL/6

MP annotation

http://metadb.riken.jp/db/bioresource_schema/brs_indicatesPhenotype

• increased sensitivity to induced morbidity/mortality
  http://purl.obolibrary.org/obo/MP_0009763

文献

http://purl.org/net/cito/citesAsAuthority

• Holger. Willenbring, et.al., Loss of p21 permits carcinogenesis from chronically damaged liver and kidney epithelial cells despite unchecked apoptosis., Cancer cell, 2008, 14
  http://rdf.ncbi.nlm.nih.gov/pubmed/18598944